James M. Joyce

Isaac Levi has long criticized causal decisiontheory on the grounds that it requiresdeliberating agents to make predictions abouttheir own actions. A rational agent cannot, heclaims, see herself as free to choose an actwhile simultaneously making a prediction abouther likelihood of performing it. Levi is wrongon both points. First, nothing in causaldecision theory forces agents to makepredictions about their own acts. Second,Levi's arguments for the ``deliberation crowdsout prediction thesis'' rely on a flawed modelof the measurement of belief. Moreover, theability of agents to adopt beliefs about theirown acts during deliberation is essentialto any plausible account of human agency andfreedom. Though these beliefs play no part inthe rationalization of actions, they arerequired to account for the causalgenesis of behavior. To explain the causes ofactions we must recognize that (a) an agentcannot see herself as entirely free in thematter of A unless she believes herdecision to perform A will cause A,and (b) she cannot come to a deliberatedecision about A unless she adoptsbeliefs about her decisions. FollowingElizabeth Anscombe and David Velleman, I arguethat an agent's beliefs about her own decisionsare self-fulfilling, and that this can beused to explain away the seeming paradoxicalfeatures of act probabilities.

I argue that one central aspect of the epistemology of causation, the use of causes as evidence for their effects, is largely independent of the metaphysics of causation. In particular, I use the formalism of Bayesian causal graphs to factor the incremental evidential impact of a cause for its effect into a direct cause-to-effect component and a backtracking component. While the “backtracking” evidence that causes provide about earlier events often obscures things, once we our restrict attention to the cause-to-effect component it is true to say promoting (inhibiting) causes raise (lower) the probabilities of their effects. This factoring assumes the same form whether causation is given an interventionist, counterfactual or probabilistic interpretation. Whether we think about causation in terms of interventions and causal graphs, counterfactuals and imaging functions, or probability raising against the background of causally homogenous partitions, if we describe the essential features of a situation correctly then the incremental evidence that a cause provides for its effect in virtue of being its cause will be the same.