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Neil Levy

University of OxfordMacquarie University
  •  Home
  •  Publications
    185
    • Most Recent
    • Most Downloaded
    • Topics
  •  Events
    16
  •  News and Updates
    165
  •  Philosophical Views

 More details
  • University of Oxford
    Regular Faculty (Part-time)
  • Macquarie University
    Department of Philosophy
    Professor (Part-time)
Monash University
Department of Philosophy
PhD, 2006
Sydney, New South Wales, Australia
0000-0002-5679-1986
Areas of Specialization
Social Epistemology
Philosophy of Psychology
Applied Ethics
Philosophy of Action
Areas of Interest
Philosophy of Action
Philosophy of Mind
Applied Ethics
Philosophy of Cognitive Science
  • All publications (185)
  •  264
    Conversation and Responsibility, by Michael McKenna (review)
    Mind 122 (486). 2013.
    Free Will and ResponsibilityResponsibility and Reactive Attitudes
  •  81
    Miller, Christian. Moral Character: An Empirical Theory.Oxford: Oxford University Press, 2013. Pp. 368. $55.00 (review)
    Ethics 124 (3): 641-645. 2014.
    Value TheoryValue Theory, Miscellaneous
  •  2
    The presumption against direct manipulation
    Neuroethics: Challenges for the 21st Century. Cambridge University Press, Cambridge. forthcoming.
    EthicsManipulation
  •  347
    Addiction is not a brain disease (and it matters)
    Frontiers in Psychiatry 4 (24): 1--7. 2013.
    The claim that addiction is a brain disease is almost universally accepted among scientists who work on addiction. The claim’s attraction rests on two grounds: the fact that addiction seems to be characterized by dysfunction in specific neural pathways and the fact that the claim seems to the compassionate response to people who are suffering. I argue that neural dysfunction is not sufficient for disease: something is a brain disease only when neural dysfunction is sufficient for impairment. I c…Read more
    The claim that addiction is a brain disease is almost universally accepted among scientists who work on addiction. The claim’s attraction rests on two grounds: the fact that addiction seems to be characterized by dysfunction in specific neural pathways and the fact that the claim seems to the compassionate response to people who are suffering. I argue that neural dysfunction is not sufficient for disease: something is a brain disease only when neural dysfunction is sufficient for impairment. I claim that the neural dysfunction that is characteristic of addiction is not sufficient for impairment, because people who suffer from that dysfunction are impaired, sufficiently to count as diseased, only given certain features of their context. Hence addiction is not a brain disease (though it is often a disease, and it may always involve brain dysfunction). I argue that accepting that addiction is not a brain disease does not entail a moralizing attitude toward people who suffer as a result of addiction; if anything, it allows for a more compassionate, and more effective, response to addiction.
    Psychiatric TaxonomyCompulsion and Addiction
  •  462
    Implicit Bias and Moral Responsibility: Probing the Data
    Philosophy and Phenomenological Research 93 (3): 3-26. 2016.
    Implicit Bias
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