Michael Baumgartner

The majority of the currently flourishing theories of actual causation are located in a broadly counterfactual framework that draws on structural equations. In order to account for cases of symmetric overdeterminiation and preemption, these theories resort to rather intricate analytical tools, most of all, to what Hitchcock has labeled explicitly nonforetracking counterfactuals. This paper introduces a regularity theoretic approach to actual causation that only employs material conditionals, standard Boolean minimization procedures, and a stability condition that regulates the behavior of causal models under model expansions. Notwithstanding its lightweight analytical toolbox, this regularity theory performs at least as well as the structural equations accounts with their heavy appliances.

This paper addresses a problem that arises when it comes to inferring deterministic causal chains from pertinent empirical data. It will be shown that to every deterministic chain there exists an empirically equivalent common cause structure. Thus, our overall conviction that deterministic chains are one of the most ubiquitous (macroscopic) causal structures is underdetermined by empirical data. It will be argued that even though the chain and its associated common cause model are empirically equivalent there exists an important asymmetry between the two models with respect to model expansions. This asymmetry might constitute a basis on which to disambiguate corresponding causal inferences on non-empirical grounds.

In "The Comparative Method" Ragin (1987) has outlined a procedure of Boolean causal reasoning operating on pure coincidence data that has meanwhile become widely known as QCA (Qualitative Comparative Analysis) among social scientists. QCA -- also in its recent form as presented in Ragin (2000) -- is designed to analyze causal structures featuring one effect and a possibly complex configuration of mutually independent direct causes of that effect. The paper at hand presents a procedure of causal reasoning that operates on the same type of empirical data as QCA and that implements Boolean techniques related to the ones resorted to by QCA, yet, in contrast to QCA, the procedure introduced here successfully identifies causal structures involving both mutually dependent causes, i.e. causal chains, and multiple effects, i.e. epiphenomena. In this sense, the paper at hand generalizes QCA.

The essential precondition of implementing interventionist techniques of causal reasoning is that particular variables are identified as so-called intervention variables. While the pertinent literature standardly brackets the question how this can be accomplished in concrete contexts of causal discovery, the first part of this paper shows that the interventionist nature of variables cannot, in principle, be established based only on an interventionist notion of causation. The second part then demonstrates that standard observational methods that draw on Bayesian networks identify intervention variables only if they also answer the questions that can be answered by interventionist techniques—which are thus rendered dispensable. The paper concludes by suggesting a way of identifying intervention variables that allows for exploiting the whole inferential potential of interventionist techniques

The first part of this paper shows that Qualitative Comparative Analysis (QCA)--also in its most recent forms as presented in Ragin (2000, 2008)--, does not correctly analyze data generated by causal chains, which, after all, are very common among causal processes in the social sciences. The incorrect modeling of data originating from chains essentially stems from QCA’s reliance on Quine-McCluskey optimization to eliminate redundancies from sufficient and necessary conditions. Baumgartner (2009a,b) has introduced a Boolean methodology, termed Coincidence Analysis (CNA), that is related to QCA, yet, contrary to the latter, does not eliminate redundancies by means of Quine-McCluskey optimization. The second part of the paper applies CNA to chain-generated data. It will turn out that CNA successfully detects causal chains in small-N data.